CLEVER detailed descriptions

This page is a sort-of FAQ for CLEVER, containing information about things that many people have asked about.

Quick introduction to the CLEVER gene header format

This is a CLEVER FASTA header:

C2|mexF|mex-6_1|ECV|CARD|AAG05882.1

The fields are delimited by ‘bars’ (|), and have the following meanings:

CLEVER version (C2) | Gene name (mexF) | CLEVER identifier (mex-6_1) | evidence codes (ECV) | source database (CARD) | Identifier in source database (AAG05882.1)

Note that the gene name often is the same as the CLEVER identifier (mex-6_1), but with a wave ‘~‘ sign added, which means that CLEVER has inferred this name, i.e. there is (as far as we know) no established gene name for this ARG. So for this gene the gene class is “mex”, the family number is 6 and the variant within the family is 1.

Evidence codes are always three letters: E or L (Established or Latent) C or M (Chromosomal or Mobile) V or P or S (Validated or Predicted or Structure-predicted)

Definition of evidence code concepts

The definitions of these concepts are as follows (see notes in italics as well!):

  • Established ARG (E): An ARG that is experimentally verified to confer antibiotic resistance and is present in human pathogens.
  • Latent ARG (L): An ARG that confers a resistance function (or is predicted to do so), but does not exist in pathogens
  • For practical reasons the current version of CLEVER (2.0) considers all ARGs present in ResFinder or CARD as “established” and all ARGs not present in those two databases as “latent”. This is intended to change for a proper pathogen definition in a future update to CLEVER.
  • Mobile ARG (M): An ARG which is present on a mobile genetic element, which could be plasmids, integrons, transposons or integrative conjugative elements.
  • Chromosomal ARG (C): Any ARG that does not meet the criteria for a mobile ARG above.
  • The current version of CLEVER (2.0) ignores anything that is not present on a plasmid in PLSDB. This is largely because of reliability issues with all other mobile genetic elements databases. We are aware that this definition may be overly conservative, but we also are wary that using other sources would introduce many false positive “mobile” ARGs. This is an obvious possible improvement for future updates to CLEVER.
  • Validated ARG (V): An ARG for which the resistance function has been verified in laboratory experiments, by showing that the presence of the gene increases the MIC of the host compared to an otherwise isogenic strain that does not carry the gene, or alternatively that over-expression of the gene induces a higher MIC compared to an isogenic reference strain.
  • Predicted ARG (P): An ARG for which its function has not been verified experimentally (see above), but has been predicted to be an ARG by fARGene (Berglund et al. 2019)
  • Structure-predicted ARG (S): An ARG for which its function has not been verified experimentally (see above), but has been predicted to be an ARG based on 3D-structure similarity by MUSTARD (Ruppé et al. 2018)
  • A small number of ARGs predicted by the Inda-Diaz et al. and Ruppé et al. 2018 papers have been experimentally verified, which is NOT yet reflected in CLEVER. This will be corrected in a future update.

ARG classes covered by CLEVER

CLEVER ClassClass nameResistance profile
aacaminoglycoside acetyltransferase (no subclass)aminoglycosides
aac2paminoglycoside N(2′)-acetyltransferaseaminoglycosides
aac3aminoglycoside N(3)-acetyltransferaseaminoglycosides
aac6paminoglycoside N(6′)-acetyltransferaseaminoglycosides
aadaminoglycoside 6-adenylyltransferaseaminoglycosides
adtaminoglycoside resistance gene (no class)aminoglycosides
antaminoglycoside O-nucleotidyltransferaseaminoglycosides
aphaminoglycoside O-phosphotransferase (no subclass)aminoglycosides
aph2baminoglycoside 2”-O-phosphotransferaseaminoglycosides
aph3baminoglycoside 3”-O-phosphotransferaseaminoglycosides
aph3paminoglycoside 3′-O-phosphotransferaseaminoglycosides
aph4aminoglycoside 4-O-phosphotransferaseaminoglycosides
aph6aminoglycoside 6-O-phosphotransferaseaminoglycosides
aph7baminoglycoside 7”-O-phosphotransferaseaminoglycosides
aph9aminoglycoside 9-O-phosphotransferaseaminoglycosides
arrrifampin ADP-ribosylating transferaserifampin
blaAclass A beta-lactamase (no subclass)beta-lactams
blaA1class A1 beta-lactamasebeta-lactams
blaB1class B1 beta-lactamasebeta-lactams
blaB3class B3 beta-lactamasebeta-lactams
blaCclass C beta-lactamase (no subclass)beta-lactams
blaC1class C1 beta-lactamasebeta-lactams
blaC2class C2 beta-lactamasebeta-lactams
blaC3class C3 beta-lactamasebeta-lactams
blaDclass D beta-lactamase (no subclass)beta-lactams
blaD1class D1 beta-lactamasebeta-lactams
blaD2class D2 beta-lactamasebeta-lactams
blaXbeta-lactamase (no class)beta-lactams
catchloramphenicol acetyltransferasechloramphenicol
cfr23S ribosomal RNA methyltransferasemacrolides,streptogramines,lincosamide
cmlchloramphenicol exporterchloramphenicol
dfrtrimethoprim-resistant dihydrofolate reductasetrimethoprims
dhrtrimethoprim-resistant dihydrofolate reductasetrimethoprims
dprsulfamethoxazole resistance genesulfonamides
ereerythromycin esterasemacrolides
ermrRNA methyltransferasemacrolides,streptogramines,lincosamide
fosfosfomycin thiol transferasefosfomycin
lnulincosamide nucleotidyltransferasemacrolides,streptogramines,lincosamide
lsaLSA ABC-F subfamily proteinmacrolides,streptogramines,lincosamide
mcrMCR phosphoethanolamine transferasepolymyxins
mecmethicillin resistant PBPbeta-lactams
mexresistance-nodulation-cell division (RND) antibiotic efflux pumpmultidrug-exporter
mphmacrolide 2′-phosphotransferasemacrolides,streptogramines,lincosamide
msrABC-F binding cassette ribosomal protection proteinmacrolides,streptogramines,lincosamide
nimnitroimidazole reductasenitroimidazoles
ompouter membrane porinbeta-lactams
oprouter membrane factor proteinmultidrug-exporter
pbppenicillin-binding proteinbeta-lactams
qnrquinolone resistance protein (qnr)quinolones
rmt16S rRNA methyltransferaseaminoglycosides
sulsulfonamide resistant dihydropteroate synthasesulfonamides
tettetracycline resistance gene (no class)tetracyclines
tetEFFtetracycline efflux pumptetracyclines
tetRPGtetracycline ribosomal protection genetetracyclines
tetXtetracycline inactivation enzymetetracyclines
trantransporter (no class)multidrug-exporter
vanvancomycin resistance genevancomycin
vatstreptogramin vat acetyltransferasemacrolides,streptogramines,lincosamide
vgaVGA ABC-F subfamily proteinmacrolides,streptogramines,lincosamide
vgbstreptogramin vgb lyasemacrolides,streptogramines,lincosamide
miscmiscellaneous ARGsvariable
uncunclassified ARGunclassified

Data sources used for CLEVER

Resistance gene databases

  • Bortolaia V, Kaas RS, Ruppe E, Roberts MC, Schwarz S, Cattoir V, et al. ResFinder 4.0 for predictions of phenotypes from genotypes. Journal of Antimicrobial Chemotherapy 2020;75:3491–500. https://doi.org/10.1093/jac/dkaa345.
  • Jia B, Raphenya AR, Alcock B, Waglechner N, Guo P, Tsang KK, et al. CARD 2017: expansion and model-centric curation of the comprehensive antibiotic resistance database. Nucleic Acids Research 2016:gkw1004. https://doi.org/10.1093/nar/gkw1004.
  • Gschwind R, Ugarcina Perovic S, Weiss M, Petitjean M, Lao J, Coelho LP, et al. ResFinderFG v2.0: a database of antibiotic resistance genes obtained by functional metagenomics. Nucleic Acids Research 2023:gkad384. https://doi.org/10.1093/nar/gkad384.

And the PLSDB plasmid database, although not technically an ARF database:

  • Schmartz GP, Hartung A, Hirsch P, Kern F, Fehlmann T, Müller R, et al. PLSDB: advancing a comprehensive database of bacterial plasmids. Nucleic Acids Research 2022;50:D273–8. https://doi.org/10.1093/nar/gkab1111.

fARGene predicted ARGs:

  • Inda-Díaz JS, Lund D, Parras-Moltó M, Johnning A, Bengtsson-Palme J, Kristiansson E. Latent antibiotic resistance genes are abundant, diverse, and mobile in human, animal, and environmental microbiomes. Microbiome. 2023;11(1):44. Published 2023 Mar 8. doi:10.1186/s40168-023-01479-0 (already present in CLEVER 1.0)
  • Victor MP, Radisic V, Grevskott DH, Marathe NP. Hospital effluent in a low-resistance setting is responsible for dissemination of novel antibiotic resistance genes into the marine environment. Ecotoxicol Environ Saf. 2025;301:118390. doi:10.1016/j.ecoenv.2025.118390
  • Victor MP, Øvreås L, Marathe NP. Characterization of known and novel clinically important antibiotic resistance genes and novel microbes from wastewater-impacted high Arctic fjord sediments. Sci Total Environ. 2025;985:179699. doi:10.1016/j.scitotenv.2025.179699
  • Li B, Jiang L, Johnson T, et al. Global health risks lurking in livestock resistome. Sci Adv. 2025;11(26):eadt8073. doi:10.1126/sciadv.adt8073 (https://zenodo.org/records/15025586)
  • Sommerville V, Meola M, Nunes-Richards A, et al. Microbial community dynamics in a traditional Swiss mountain cheese over 142 years of cheesemaking. bioRxiv, 2026.02.26.708305 (2026). doi:10.64898/2026.02.26.708305

MUSTARD/PCM predicted ARGs:

  • Ruppé E, Ghozlane A, Tap J, et al. Prediction of the intestinal resistome by a three-dimensional structure-based method. Nat Microbiol. 2019;4(1):112-123. doi:10.1038/s41564-018-0292-6
  • Wang K, Xu J, Li X, et al. Evolutionary selection of trimethoprim-resistant dfrA genes in lytic phages affects phage and host fitness during infection. Sci Adv. 2025;11(39):eadt4817. doi:10.1126/sciadv.adt4817